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Reprint of: Role of enteric neurotransmission in host defense and protection of the gastrointestinal tract.

Autonomic neuroscience : basic & clinical

Sharkey KA, Savidge TC.
PMID: 24674836
Auton Neurosci. 2014 May;182:70-82. doi: 10.1016/j.autneu.2014.03.004. Epub 2014 Mar 15.

Host defense is a vital role played by the gastrointestinal tract. As host to an enormous and diverse microbiome, the gut has evolved an elaborate array of chemical and physicals barriers that allow the digestion and absorption of nutrients...

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Sharkey KA, Savidge TC. Reprint of: Role of enteric neurotransmission in host defense and protection of the gastrointestinal tract. Auton Neurosci. 2014;182:70-82doi: 10.1016/j.autneu.2014.03.004.
Sharkey, K. A., & Savidge, T. C. (2014). Reprint of: Role of enteric neurotransmission in host defense and protection of the gastrointestinal tract. Autonomic neuroscience : basic & clinical, 18270-82. https://doi.org/10.1016/j.autneu.2014.03.004
Sharkey, Keith A, and Savidge, Tor C. "Reprint of: Role of enteric neurotransmission in host defense and protection of the gastrointestinal tract." Autonomic neuroscience : basic & clinical vol. 182 (2014): 70-82. doi: https://doi.org/10.1016/j.autneu.2014.03.004
Sharkey KA, Savidge TC. Reprint of: Role of enteric neurotransmission in host defense and protection of the gastrointestinal tract. Auton Neurosci. 2014 May;182:70-82. doi: 10.1016/j.autneu.2014.03.004. Epub 2014 Mar 15. PMID: 24674836.
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Driving restrictions in patients following syncope is difficult for physicians.

Autonomic neuroscience : basic & clinical

Raj SR.
PMID: 19850538
Auton Neurosci. 2009 Dec 03;151(2):71-3. doi: 10.1016/j.autneu.2009.09.024. Epub 2009 Oct 21.

Syncope occurring while driving has obvious implications for personal and public safety. In this report, the authors aimed to define the clinical characteristics, causes, and prognosis of syncope while driving. Using a case-control design, they studied consecutive patients evaluated...

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Raj SR. Driving restrictions in patients following syncope is difficult for physicians. Auton Neurosci. 2009;151(2):71-3doi: 10.1016/j.autneu.2009.09.024.
Raj, S. R. (2009). Driving restrictions in patients following syncope is difficult for physicians. Autonomic neuroscience : basic & clinical, 151(2), 71-3. https://doi.org/10.1016/j.autneu.2009.09.024
Raj, Satish R. "Driving restrictions in patients following syncope is difficult for physicians." Autonomic neuroscience : basic & clinical vol. 151,2 (2009): 71-3. doi: https://doi.org/10.1016/j.autneu.2009.09.024
Raj SR. Driving restrictions in patients following syncope is difficult for physicians. Auton Neurosci. 2009 Dec 03;151(2):71-3. doi: 10.1016/j.autneu.2009.09.024. Epub 2009 Oct 21. PMID: 19850538.
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Highlights in clinical autonomic neuroscience: latest developments in the early detection of diabetic autonomic neuropathy.

Autonomic neuroscience : basic & clinical

Cheshire WP, Freeman R.
PMID: 22306250
Auton Neurosci. 2012 Apr 03;167(1):4-6. doi: 10.1016/j.autneu.2012.01.008. Epub 2012 Feb 10.

No abstract available.

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Cheshire WP, Freeman R. Highlights in clinical autonomic neuroscience: latest developments in the early detection of diabetic autonomic neuropathy. Auton Neurosci. 2012;167(1):4-6doi: 10.1016/j.autneu.2012.01.008.
Cheshire, W. P., & Freeman, R. (2012). Highlights in clinical autonomic neuroscience: latest developments in the early detection of diabetic autonomic neuropathy. Autonomic neuroscience : basic & clinical, 167(1), 4-6. https://doi.org/10.1016/j.autneu.2012.01.008
Cheshire, William P, and Freeman, Roy. "Highlights in clinical autonomic neuroscience: latest developments in the early detection of diabetic autonomic neuropathy." Autonomic neuroscience : basic & clinical vol. 167,1 (2012): 4-6. doi: https://doi.org/10.1016/j.autneu.2012.01.008
Cheshire WP, Freeman R. Highlights in clinical autonomic neuroscience: latest developments in the early detection of diabetic autonomic neuropathy. Auton Neurosci. 2012 Apr 03;167(1):4-6. doi: 10.1016/j.autneu.2012.01.008. Epub 2012 Feb 10. PMID: 22306250.
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Highlights in basic autonomic neurosciences: central adenosine A1 receptor - the key to a hypometabolic state and therapeutic hypothermia?.

Autonomic neuroscience : basic & clinical

Tupone D, Madden CJ, Morrison SF.
PMID: 23465354
Auton Neurosci. 2013 Jun;176(1):1-2. doi: 10.1016/j.autneu.2013.02.004. Epub 2013 Mar 01.

The positive outcome that hypothermia contributes to brain and cardiac protection following ischemia has stimulated research in the development of pharmacological approaches to induce a hypothermic/hypometabolic state. Here we review three papers to highlight the role of the adenosine...

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Tupone D, Madden CJ, Morrison SF. Highlights in basic autonomic neurosciences: central adenosine A1 receptor - the key to a hypometabolic state and therapeutic hypothermia?. Auton Neurosci. 2013;176(1):1-2doi: 10.1016/j.autneu.2013.02.004.
Tupone, D., Madden, C. J., & Morrison, S. F. (2013). Highlights in basic autonomic neurosciences: central adenosine A1 receptor - the key to a hypometabolic state and therapeutic hypothermia?. Autonomic neuroscience : basic & clinical, 176(1), 1-2. https://doi.org/10.1016/j.autneu.2013.02.004
Tupone, D, et al. "Highlights in basic autonomic neurosciences: central adenosine A1 receptor - the key to a hypometabolic state and therapeutic hypothermia?." Autonomic neuroscience : basic & clinical vol. 176,1 (2013): 1-2. doi: https://doi.org/10.1016/j.autneu.2013.02.004
Tupone D, Madden CJ, Morrison SF. Highlights in basic autonomic neurosciences: central adenosine A1 receptor - the key to a hypometabolic state and therapeutic hypothermia?. Auton Neurosci. 2013 Jun;176(1):1-2. doi: 10.1016/j.autneu.2013.02.004. Epub 2013 Mar 01. PMID: 23465354; PMCID: PMC4990001.
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Highlights in clinical autonomic neurosciences: device-based therapy for resistant hypertension.

Autonomic neuroscience : basic & clinical

Raj SR.
PMID: 22137183
Auton Neurosci. 2012 Jan 26;166(1):1-3. doi: 10.1016/j.autneu.2011.11.003. Epub 2011 Dec 01.

No abstract available.

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Raj SR. Highlights in clinical autonomic neurosciences: device-based therapy for resistant hypertension. Auton Neurosci. 2011;166(1):1-3doi: 10.1016/j.autneu.2011.11.003.
Raj, S. R. (2012). Highlights in clinical autonomic neurosciences: device-based therapy for resistant hypertension. Autonomic neuroscience : basic & clinical, 166(1), 1-3. https://doi.org/10.1016/j.autneu.2011.11.003
Raj, Satish R. "Highlights in clinical autonomic neurosciences: device-based therapy for resistant hypertension." Autonomic neuroscience : basic & clinical vol. 166,1 (2012): 1-3. doi: https://doi.org/10.1016/j.autneu.2011.11.003
Raj SR. Highlights in clinical autonomic neurosciences: device-based therapy for resistant hypertension. Auton Neurosci. 2012 Jan 26;166(1):1-3. doi: 10.1016/j.autneu.2011.11.003. Epub 2011 Dec 01. PMID: 22137183.
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Risk factors and clinical features of paroxysmal sympathetic hyperactivity after spontaneous intracerebral hemorrhage.

Autonomic neuroscience : basic & clinical

Li Z, Chen W, Zhu Y, Han K, Wang J, Chen J, Zhang D, Yu M, Lv L, Hou L.
PMID: 32097879
Auton Neurosci. 2020 Jan 27;225:102643. doi: 10.1016/j.autneu.2020.102643. Epub 2020 Jan 27.

BACKGROUND AND PURPOSE: Paroxysmal sympathetic hyperactivity (PSH) is a rare complication of spontaneous intracerebral hemorrhage (ICH).We aimed to evaluate the risk factors and clinical features for PSH after ICH.METHODS: From January 1, 2013 to April 1, 2018, patients with...

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Li Z, Chen W, Zhu Y, et al. Risk factors and clinical features of paroxysmal sympathetic hyperactivity after spontaneous intracerebral hemorrhage. Auton Neurosci. 2020;225:102643doi: 10.1016/j.autneu.2020.102643.
Li, Z., Chen, W., Zhu, Y., Han, K., Wang, J., Chen, J., Zhang, D., Yu, M., Lv, L., & Hou, L. (2020). Risk factors and clinical features of paroxysmal sympathetic hyperactivity after spontaneous intracerebral hemorrhage. Autonomic neuroscience : basic & clinical, 225102643. https://doi.org/10.1016/j.autneu.2020.102643
Li, Zhenxing, et al. "Risk factors and clinical features of paroxysmal sympathetic hyperactivity after spontaneous intracerebral hemorrhage." Autonomic neuroscience : basic & clinical vol. 225 (2020): 102643. doi: https://doi.org/10.1016/j.autneu.2020.102643
Li Z, Chen W, Zhu Y, Han K, Wang J, Chen J, Zhang D, Yu M, Lv L, Hou L. Risk factors and clinical features of paroxysmal sympathetic hyperactivity after spontaneous intracerebral hemorrhage. Auton Neurosci. 2020 Jan 27;225:102643. doi: 10.1016/j.autneu.2020.102643. Epub 2020 Jan 27. PMID: 32097879.
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Threshold and saturation pressures of baroreflex-mediated myocardial interstitial acetylcholine release in rats.

Autonomic neuroscience : basic & clinical

Kawada T, Sata Y, Akiyama T, Shimizu S, Sonobe T, Pearson JT, Sugimachi M.
PMID: 32097880
Auton Neurosci. 2020 Feb 19;225:102657. doi: 10.1016/j.autneu.2020.102657. Epub 2020 Feb 19.

Cardiac microdialysis allows the assessment of cardiac efferent vagal nerve activity from myocardial interstitial acetylcholine (ACh) levels with minimal influence on the neural control of the heart; however, a total picture of the baroreflex-mediated myocardial interstitial ACh release including...

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Kawada T, Sata Y, Akiyama T, et al. Threshold and saturation pressures of baroreflex-mediated myocardial interstitial acetylcholine release in rats. Auton Neurosci. 2020;225:102657doi: 10.1016/j.autneu.2020.102657.
Kawada, T., Sata, Y., Akiyama, T., Shimizu, S., Sonobe, T., Pearson, J. T., & Sugimachi, M. (2020). Threshold and saturation pressures of baroreflex-mediated myocardial interstitial acetylcholine release in rats. Autonomic neuroscience : basic & clinical, 225102657. https://doi.org/10.1016/j.autneu.2020.102657
Kawada, Toru, et al. "Threshold and saturation pressures of baroreflex-mediated myocardial interstitial acetylcholine release in rats." Autonomic neuroscience : basic & clinical vol. 225 (2020): 102657. doi: https://doi.org/10.1016/j.autneu.2020.102657
Kawada T, Sata Y, Akiyama T, Shimizu S, Sonobe T, Pearson JT, Sugimachi M. Threshold and saturation pressures of baroreflex-mediated myocardial interstitial acetylcholine release in rats. Auton Neurosci. 2020 Feb 19;225:102657. doi: 10.1016/j.autneu.2020.102657. Epub 2020 Feb 19. PMID: 32097880.
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Attenuated baroreflex in a Parkinson's disease animal model coincides with impaired activation of non-C1 neurons.

Autonomic neuroscience : basic & clinical

Cabral LM, Moreira TS, Takakura AC, Falquetto B.
PMID: 32092676
Auton Neurosci. 2020 Feb 15;225:102655. doi: 10.1016/j.autneu.2020.102655. Epub 2020 Feb 15.

Orthostatic hypotension is one of the most common symptoms observed in Parkinson's disease (PD), a neurodegenerative disease caused by death of dopaminergic neurons in the substantia nigra pars compacta (SNc), and it is associated with denervation of the heart...

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Cabral LM, Moreira TS, Takakura AC, et al. Attenuated baroreflex in a Parkinson's disease animal model coincides with impaired activation of non-C1 neurons. Auton Neurosci. 2020;225:102655doi: 10.1016/j.autneu.2020.102655.
Cabral, L. M., Moreira, T. S., Takakura, A. C., & Falquetto, B. (2020). Attenuated baroreflex in a Parkinson's disease animal model coincides with impaired activation of non-C1 neurons. Autonomic neuroscience : basic & clinical, 225102655. https://doi.org/10.1016/j.autneu.2020.102655
Cabral, Laís M, et al. "Attenuated baroreflex in a Parkinson's disease animal model coincides with impaired activation of non-C1 neurons." Autonomic neuroscience : basic & clinical vol. 225 (2020): 102655. doi: https://doi.org/10.1016/j.autneu.2020.102655
Cabral LM, Moreira TS, Takakura AC, Falquetto B. Attenuated baroreflex in a Parkinson's disease animal model coincides with impaired activation of non-C1 neurons. Auton Neurosci. 2020 Feb 15;225:102655. doi: 10.1016/j.autneu.2020.102655. Epub 2020 Feb 15. PMID: 32092676.
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Real-world droxidopa or midodrine treatment persistence in patients with neurogenic orthostatic hypotension or orthostatic hypotension.

Autonomic neuroscience : basic & clinical

Kymes SM, Sullivan C, Jackson K, Raj SR.
PMID: 32200263
Auton Neurosci. 2020 Feb 28;225:102659. doi: 10.1016/j.autneu.2020.102659. Epub 2020 Feb 28.

BACKGROUND: Neurogenic orthostatic hypotension (nOH) is a subtype of orthostatic hypotension (OH) observed in the presence of neuropathy and is associated with increased risk of falling, impaired function, and poor quality of life. Droxidopa and midodrine are approved in...

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Kymes SM, Sullivan C, Jackson K, et al. Real-world droxidopa or midodrine treatment persistence in patients with neurogenic orthostatic hypotension or orthostatic hypotension. Auton Neurosci. 2020;225:102659doi: 10.1016/j.autneu.2020.102659.
Kymes, S. M., Sullivan, C., Jackson, K., & Raj, S. R. (2020). Real-world droxidopa or midodrine treatment persistence in patients with neurogenic orthostatic hypotension or orthostatic hypotension. Autonomic neuroscience : basic & clinical, 225102659. https://doi.org/10.1016/j.autneu.2020.102659
Kymes, Steven M, et al. "Real-world droxidopa or midodrine treatment persistence in patients with neurogenic orthostatic hypotension or orthostatic hypotension." Autonomic neuroscience : basic & clinical vol. 225 (2020): 102659. doi: https://doi.org/10.1016/j.autneu.2020.102659
Kymes SM, Sullivan C, Jackson K, Raj SR. Real-world droxidopa or midodrine treatment persistence in patients with neurogenic orthostatic hypotension or orthostatic hypotension. Auton Neurosci. 2020 Feb 28;225:102659. doi: 10.1016/j.autneu.2020.102659. Epub 2020 Feb 28. PMID: 32200263.
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Regulation of acute reflectory hyperinflammation in viral and other diseases by means of stellate ganglion block. A conceptual view with a focus on Covid-19.

Autonomic neuroscience : basic & clinical

Fischer L, Barop H, Ludin SM, Schaible HG.
PMID: 34894589
Auton Neurosci. 2021 Nov 10;237:102903. doi: 10.1016/j.autneu.2021.102903. Epub 2021 Nov 10.

Whereas the autonomic nervous system (ANS) and the immune system used to be assigned separate functions, it has now become clear that the ANS and the immune system (and thereby inflammatory cascades) work closely together. During an acute immune...

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Fischer L, Barop H, Ludin SM, et al. Regulation of acute reflectory hyperinflammation in viral and other diseases by means of stellate ganglion block. A conceptual view with a focus on Covid-19. Auton Neurosci. 2021;237:102903doi: 10.1016/j.autneu.2021.102903.
Fischer, L., Barop, H., Ludin, S. M., & Schaible, H. G. (2021). Regulation of acute reflectory hyperinflammation in viral and other diseases by means of stellate ganglion block. A conceptual view with a focus on Covid-19. Autonomic neuroscience : basic & clinical, 237102903. https://doi.org/10.1016/j.autneu.2021.102903
Fischer, Lorenz, et al. "Regulation of acute reflectory hyperinflammation in viral and other diseases by means of stellate ganglion block. A conceptual view with a focus on Covid-19." Autonomic neuroscience : basic & clinical vol. 237 (2021): 102903. doi: https://doi.org/10.1016/j.autneu.2021.102903
Fischer L, Barop H, Ludin SM, Schaible HG. Regulation of acute reflectory hyperinflammation in viral and other diseases by means of stellate ganglion block. A conceptual view with a focus on Covid-19. Auton Neurosci. 2021 Nov 10;237:102903. doi: 10.1016/j.autneu.2021.102903. Epub 2021 Nov 10. PMID: 34894589.
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Autonomic dysfunction and cardiovascular risk in post-traumatic stress disorder.

Autonomic neuroscience : basic & clinical

Fu Q.
PMID: 34844132
Auton Neurosci. 2021 Nov 19;237:102923. doi: 10.1016/j.autneu.2021.102923. Epub 2021 Nov 19.

BACKGROUND: Patients with post-traumatic stress disorder (PTSD) have an increased risk for cardiovascular disease. The underlying mechanisms are unclear but impaired autonomic function may contribute. However, research in this field has shown contradictory results and the causal links between...

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Fu Q. Autonomic dysfunction and cardiovascular risk in post-traumatic stress disorder. Auton Neurosci. 2021;237:102923doi: 10.1016/j.autneu.2021.102923.
Fu, Q. (2021). Autonomic dysfunction and cardiovascular risk in post-traumatic stress disorder. Autonomic neuroscience : basic & clinical, 237102923. https://doi.org/10.1016/j.autneu.2021.102923
Fu, Qi. "Autonomic dysfunction and cardiovascular risk in post-traumatic stress disorder." Autonomic neuroscience : basic & clinical vol. 237 (2021): 102923. doi: https://doi.org/10.1016/j.autneu.2021.102923
Fu Q. Autonomic dysfunction and cardiovascular risk in post-traumatic stress disorder. Auton Neurosci. 2021 Nov 19;237:102923. doi: 10.1016/j.autneu.2021.102923. Epub 2021 Nov 19. PMID: 34844132.
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Severe dysautonomia in glycine receptor antibody-positive progressive encephalomyelitis with rigidity and myoclonus (PERM): A case report.

Autonomic neuroscience : basic & clinical

Mizutani Y, Adachi S, Nakano S, Hayashi K, Higashi A, Kikuchi K, Maeda T, Murate K, Shima S, Iizuka T, Ueda A, Ito M, Watanabe H.
PMID: 34801829
Auton Neurosci. 2021 Nov 16;237:102910. doi: 10.1016/j.autneu.2021.102910. Epub 2021 Nov 16.

Progressive encephalomyelitis with rigidity and myoclonus (PERM) is a severe form of stiff-person spectrum disorder. We report a 59-year-old man who presented with progressive encephalomyelitis causing diplopia, bulbar palsy, severe dysautonomia, followed by stiffness and myoclonic cluster. Laboratory tests...

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Mizutani Y, Adachi S, Nakano S, et al. Severe dysautonomia in glycine receptor antibody-positive progressive encephalomyelitis with rigidity and myoclonus (PERM): A case report. Auton Neurosci. 2021;237:102910doi: 10.1016/j.autneu.2021.102910.
Mizutani, Y., Adachi, S., Nakano, S., Hayashi, K., Higashi, A., Kikuchi, K., Maeda, T., Murate, K., Shima, S., Iizuka, T., Ueda, A., Ito, M., & Watanabe, H. (2021). Severe dysautonomia in glycine receptor antibody-positive progressive encephalomyelitis with rigidity and myoclonus (PERM): A case report. Autonomic neuroscience : basic & clinical, 237102910. https://doi.org/10.1016/j.autneu.2021.102910
Mizutani, Yasuaki, et al. "Severe dysautonomia in glycine receptor antibody-positive progressive encephalomyelitis with rigidity and myoclonus (PERM): A case report." Autonomic neuroscience : basic & clinical vol. 237 (2021): 102910. doi: https://doi.org/10.1016/j.autneu.2021.102910
Mizutani Y, Adachi S, Nakano S, Hayashi K, Higashi A, Kikuchi K, Maeda T, Murate K, Shima S, Iizuka T, Ueda A, Ito M, Watanabe H. Severe dysautonomia in glycine receptor antibody-positive progressive encephalomyelitis with rigidity and myoclonus (PERM): A case report. Auton Neurosci. 2021 Nov 16;237:102910. doi: 10.1016/j.autneu.2021.102910. Epub 2021 Nov 16. PMID: 34801829.
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Showing 1 to 12 of 233 entries